Covid’s Continual Assault on the Kidneys
by David Archibald
27 February 2026
The Epstein files tell us that, in 2011, JP Morgan sought Epstein’s advice on how to structure an investment product offering. Epstein advised that the products offered should include one solely devoted to vaccines. The investment company of Rishi Sunak, now a former UK Prime Minister, also decided to invest in vaccines in 2011, taking a position in Moderna. They must have been confident that the planned pandemic was well in train. Hunter Biden followed with an investment in Metabiota in 2014.
Another year passes and in 2015 Ralph Baric, the scientific brains behind covid at the University of North Carolina, Chapel Hill, shipped transgenic mice with humanised ACE2 receptors to Batwoman’s lab in Wuhan.
From that we can imply that covid’s method of infection, the covid virion entering cells via the ACE2 receptor, had been chosen by 2015 at the latest. Baric was so confident of his creation that he published a paper that year predicting a viral outbreak. It was entitled “A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence.”
It is likely that it was he, Baric, who chose a coronavirus to be the vector for the planned pandemic. In 1992 he published a paper entitled An Experimental Model for Myocarditis and Congestive Heart Failure after Rabbit Coronavirus Infection. Why a coronavirus? This is likely because it is well known amongst virologists that you can’t make a vaccine for a coronavirus because they mutate too fast. Baric didn’t want to make money, he just wanted to kill people. He probably didn’t want to have the potential for his creation to be thwarted by a vaccine.
Why heart disease instead of cancer or some other type of affliction? This choice was likely inspired by the track record of the Spanish Flu outbreak of 1918, which caused an elevated incidence of heart attacks into the 1970s. The decline in the heart attack rate from then wasn’t due to better treatment, it was the last of the cohort from 1918 dying off due to old age.
There was another consequence of the difficulty of developing a vaccine for a coronavirus. The people who ran the covid program in the National Institutes of Health (NIH) in the US, Anthony Fauci and his boss Francis Collins, had structured things so that they received a chunk of the royalty payments that Pfizer and Moderna paid to license the covid vaccine developed by the NIH. So, Fauci didn’t retire until 2022 at the age of 82 and Collins didn’t retire until 2025 at the age of 74. Anecdotally, Fauci’s payment from this was US$9 million.
So how did the vaccines go? It turns out that the more covid shots you took, the sicker you became. The vaccinated were more likely to be infected than the unvaccinated and this effect increased with more shots from the third dose onwards. This was due to a changed immune response that treated pathogens as if they were something as harmless as pollen. The vaccinated also shed a lot of spike protein that had been created in their bodies. We know this from the incidence of women experiencing menstrual arrhythmia from being in proximity to the vaccinated.
And now we have the results of a South Korean study of 8.4 million people: 1-year risks of cancers associated with COVID-19 vaccination: a large population-based cohort study in South Korea. The study found that the incidence of thyroid cancer increased by 35% following vaccination, prostate cancer by 69%, colorectal cancer by 28%, lung cancer by 53% and so on. There isn’t an organ in body that isn’t affected by covid to some extent.
For example, the incidence of Achilles tendon injuries in the NFL has doubled since 2020. This is because covid attacks connective tissue.
We are told that medical science is puzzled by the increased incidence of colon cancer in young people since 2020. The increase is explained by the fact that Ralph Baric chose the ACE2 receptor for his creation’s entry into cells. The amount of ACE2 receptors by organ varies as tabled here:
The first column is the weight of ACE2 receptors in millionths of a gram per gram of total protein in the organ. The second column is the weight of the organ. From the combination of those two, we get the total weight of ACE2 receptors per organ. ACE2 receptor expression drops away rapidly after the gallbladder.
The next thing to consider is the blood flow through each organ. For example, the kidneys get a quarter of the blood flow from the heart. Up to this point the small intestine should be getting about three times the spike protein-caused inflammation of the kidneys and colon. Beyond that there is a big unknown, which is that the kidneys filter things out of the blood while the other organs add things to it. So the kidneys could be the biggest target for the spike protein, from either vaccination or live virus. The liver also filters, but its ACE2 receptor expression is one eighth that of the kidneys, weight for weight.
That’s our theory. Is there any evidence that it is correct? Yes, it turns out that our kidneys are taking a whacking:
The graph above shows the annual incidence of hospital treatment for chronic kidney disease in the National Health Service (NHS) of the UK from 2013 to 2025. There has been a 50% increase from the pre-2020 trend. The intestines have also had a 50% increase since covid was thrust upon us:
Another thing about covid is that it suppresses the immune system so that other diseases, in the body but held in check by the immune system normally, start to run rampant. This chart shows that the incidence of liver disease with hepatitis has tripled in the NHS since covid came along:
Just because the incidence rate of a disease has tripled doesn’t mean it stops there. These sorts of diseases are caused by cumulative inflammatory insult and we are just at the beginning of that process. There are plenty of other indications of covid’s march through the organs. From the NHS again:
Endometriosis is three times higher than it was pre-covid. This is a big signal affecting fertility.
The left-hand graph shows that so far there has only been a doubling of the rate of incipient cervical cancers in the UK, while the right-hand graph shows there is a non-trivial signal in the rate of malignant neoplasms of the penis in the over-70 cohort.
The incidence of scabies in the UK has tripled over the last three years as covid suppresses the immune system.
This graph is a stronger signal that something has gone wrong in the incidence of tuberculosis, up seven-fold so far.
This chart is of amputation of limbs in the NHS of the UK, 2013 to 2025. Covid is fibrotic and causes blood clots in minor capillaries. This reduces circulation with the potential for gangrene to follow, and the necessity for amputation after that. So far this is up three-fold in the UK.
Whooping cough in the NHS system of England is up at least five-fold in the last two years.
These charts, from Finland on the left and England on the right, are of the incidence of congenital abnormalities caused by covid during pregnancy. Pregnant women should spare no effort in avoiding covid. As well as the congenital abnormalities, children with in-utero covid exposure showed altered brain structures at birth and lower cognitive/social scores by age two, with 52% at high-risk for developmental delays.
This chart is of quarterly sales of the anti-Alzheimers drug donepezil in Australia from 2015 to 2025. The pre-covid period is the yellow on the left and post-covid is the red on the right. Sales have tripled from 2021.
This graph is of deaths in the US of school-age children, 5 – 17, from 2010 to 2025. Deaths in this cohort are up 20% since 2021.
And from Sweden, this graph is of the causes of death of children aged 10 to 14 years, from 1997 to 2024. There has been an 89% annual growth rate over the last two years. At that rate deaths will be up ten-fold in 3.6 years. Key areas of growth among deaths of 10-14 year olds are:
- neurological / congenital CNS / brain disorders
- infections / sepsis / pneumonia / respiratory
- rare cancers / haematology / immune
- cardiac / vascular / circulatory.
There is no dominant block. In other words, this suggests a systemic, heterogeneous increase, as opposed to one epidemic or one disease class.
From the US, this graph shows the amount of influenza-A virus in sampling at 181 wastewater stations across the country. The monotonic increase in peak annual viral load from influenza is what you would expect from ongoing weakening of immunity.
The consequence of continuing inflammation from circulating spike protein, and covid virions chewing on the CD8 cells of the immune system, is declining health of the workforce. This is a graph of the number of people in the US workforce with a disability from 2008 to 2025. It held pretty steady up to 2021 when covid vaccination took off. The number of disabled workers in the US is now rising at about one million per annum. Until the underlying cause of the disablement is stopped, the uptrend won’t stop.
We are told that Australia is in a ‘baby drought’ but we aren’t told why or by how much:
Taiwan has a population of 26 million, which is pretty close to Australia’s. Taiwan does a far better job of releasing birth and death statistics. The following graph is of monthly births and deaths in Taiwan from 2019 to 2025. It is the best indication we can get of what is likely happening in Australia.
Taiwan was a happy place in which births and deaths equaled each other at about 14,000 per month. That was until covid came along. Deaths are now running at 16,000 per month. The birth rate has entered a decline, now down to about 8,000 per month. So net population loss is 8,000 per month currently, translating to 100,000 per annum.
How much of the decline in births is due to miscarriages and how much is due to reduced fertility is unknown. It could be that persistent covid infection is affecting the ovarian reserve, quantified by the level of anti-Mullerian hormone (AMH). AMH is a glycoprotein produced by ovarian follicles. The AMH level is a predictor of ovarian reserve and thus the ability to conceive. In early 2024 a paper was published that found that that a covid infection, on average, reduced a woman’s AMH level by 0.24 ng/ml. This is a big deal because a woman’s peak fertility is at the age of 18 with an AMH level of 6 ng/ml. Which means that, combined with natural decline, a total of only 12 covid infections will make her completely infertile by her early thirties. In the current age that is also when a lot of women are deciding to start families. How that works out is shown in the following graph:
The blue line is the natural decline from the age of 18. AMH drops to a low level in the early forties, after which it becomes increasing difficult to conceive. The red line is the modelled effect of annual covid infections from the age of 20. The green line is the same effect from the age of 30. Our society is likely to find that babies and annual covid infections are mutually exclusive. Some girls, with many covid infections during childhood, may not even get to the start line. This is an existential issue.
There is one matter that the Epstein files cleared up, by its absence. Why would the covid conspirators unleash covid on humanity when it would mean that their own children would suffer from this plague? There is no mention in the Epstein files of a drug that could be taken to stop the disease, in the manner of Truvada and Biktarvy for the HIV-afflicted. It seems that they actually thought that the covid vaccines would work, even though the impossibility of a vaccine for a coronavirus is taught in Virology 101. Perhaps they were blinded by the money they planned to make out of Pfizer and Moderna.
Pfizer did cobble together an antiviral for covid, called Paxlovid, but it seems to have been designed to maximise profit rather than be used as a long-term treatment or as a prophylactic. You can only use it for five days at a time due to its potential to cause liver damage.
There is a solution based on currently known science. I have assembled a formulation which would approach Paxlovid in antiviral efficacy, milligram for milligram, but without the side effects. It could be taken indefinitely which is necessary to suppress viral infections in immunoprotected tissues, as it is with HIV. How it would work is illustrated by the following diagram of how covid replicates:
An individual covid virion is quite small, weighing one femtogram (a billionth of a millionth of a gram). The virion passes from the intercellular fluid through the ACE2 receptor on the cell membrane and into the intracellular fluid. Once there it assembles two polyproteins — pp1a and pp1ab. These are cleaved into 16 non-structural proteins, nsp for short.
One of these, nsp3, becomes the protease PLpro. Another, nsp5, becomes the protease Mpro. These two proteases do the cleaving of the polyproteins. Another non-structural protein, nsp12, becomes the polymerase RdRp. In theory, if you block any one of these, viral replication will stop and you will suppress the viral infection enough to have a normal life. Also in theory, the more proteases and polymerases you block simultaneously, the greater the synergy achieved between the molecules of your drug.
In Paxlovid, the molecule nirmatrelvir blocks Mpro. By itself, nirmatrelvir doesn’t last long in the body because it is rapidly cleared by the liver. To slow down clearance, it is combined with a molecule called ritonavir which is a CYP3A4 inhibitor. CYP3A4 is one of the liver’s clearance pathways.
By comparison, the new formulation, based on open-source science, will derive synergy from blocking PLpro, Mpro and RdRp simultaneously, aided by inhibition of a couple of subsidiary viral replication pathways. None of the molecules involved have any toxicity to speak of and so the whole formulation will be nontoxic. Proving up and optimising the formulation will require some in vitro and in vivo work.
In the interim, while that work is undertaken, possibly hundreds of Australian women may be made infertile each day due to repeated covid infections. Time is of the essence.
David Archibald is the author of The Anticancer Garden in Australia.
P.S. The author has produced a cancer protocol which has been successful in a number of prostate cancer cases. In one of these, the protocol provided two successful outcomes as illustrated by this graph:
Initially the patient was on the protocol for three weeks and achieved a reduction in his PSA level from 9 to 6 ng/ml, a fall of 1 ng/ml/week. The patient opted to proceed with a scheduled prostatectomy. The first PSA test after the prostatectomy had a result of zero PSA in his blood, as it should be. The third PSA test after the prostatectomy had a result of 0.11 ng/ml and thereafter the doubling time of his PSA level was three months. It seems that the surgery of the prostatectomy had released some prostate cancer stem cells from the prostate capsule. The patient went back on the protocol, and with good compliance, is now recording results of 0.01 ng/ml. This is the happy circumstance of one patient providing two successful results — an inhibitory effect on prostate cancer and clearance of metastatic prostate cancer. Based on my experience and the current state of covid science, treating covid will be of the same order of difficulty as treating metastatic prostate cancer, and is therefore achievable.